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A medium size, non-sterile, chalazion clamp.
Self-retaining with discoid ends; used to hold and prevent a chalazion from bleeding during its surgery.
Chalazion is a chronic noninfective inflammation of the sebaceous glands of the eyelid.
They commonly affect the meibomian glands in the tarsal plate, resulting clinically as a painless, firm nodule of the eyelid.
Marginal chalazia are caused by inflammation of the gland of Zeis located at the lid margin. They can affect both the upper and lower lids.
Acne rosacea and posterior blepharitis are commonly associated with chalazion. Hyperimmunoglobulinemia E (Job syndrome) can be associated with aggressive chalazion.
Meibomitis predisposes to chalazion formation. Meibomitis causes blockage of the meibomian orifices and clogging of the glands with secretions.
This result in swellings in the eyelid called chalazion and hardening of secretions with time. Chalazion is a noninfective condition.
However, it can become infected and acutely inflamed, causing a tense, warm lid swelling called hordeolum internum.
This is how meibomitis is linked to the pathogenesis of chalazion formation and recurrence.
Histologically, deep dermal or subcutaneous suppurative lipogranulomatous inflammation exists, containing neutrophils, plasma cells, lymphocytes, histiocytes, and giant cells in a zonal configuration around central lipid material. A pseudocapsule surrounds the cellular infiltrate.[1] Many chalazia resolve within 2 weeks of a topical antibiotic and steroid medication and application of warm compresses.[2] These aid in reducing inflammation and increasing the local blood supply.
Incision and curettage is a conventional and effective treatment of chalazion
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